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Results: 6
Number of items: 6
  • van der Harg, J. M., Eggels, L., Bangel, F. N., Ruigrok, S. R., Zwart, R., Hoozemans, J. J. M., la Fleur, S. E., & Scheper, W. (2017). Insulin deficiency results in reversible protein kinase A activation and tau phosphorylation. Neurobiology of Disease, 103, 163-173. https://doi.org/10.1016/j.nbd.2017.04.005
  • Open Access
    van der Harg, J. M. (2017). Tau phosphorylation as adaptive response to metabolic dysfunction in the brain. [Thesis, fully internal, Universiteit van Amsterdam].
  • Open Access
    Elfrink, H. L. (2014). Rab6 and unfolded protein response-mediated proteostasis in Alzheimer’s disease. [Thesis, fully internal, Universiteit van Amsterdam].
  • Open Access
    Nijholt, A. T. (2012). The unfolded protein response: a common pathomechanism in tauopathies. [Thesis, fully internal, Universiteit van Amsterdam]. Boxpress.
  • Open Access
    Chafekar, S. M. (2008). The aggregation state of β-amyloid in Alzheimer’s Disease. [Thesis, fully internal, Universiteit van Amsterdam].
  • Open Access
    Hoozemans, J. J. M., van Haastert, E. S., Veerhuis, R., Arendt, T., Scheper, W., Eikelenboom, P., & Rozemuller, A. J. M. (2005). Maximal COX-2 and ppRb expression in neurons occurs during early Braak stages prior to the maximal activation of astrocytes and microglia in Alzheimer's disease. Nederlands Tijdschrift voor Geneeskunde, 2, 27.
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